Anti-TFAP2B Polyclonal Antibody-一抗-抗体-蛋白与免疫

Anti-TFAP2B Polyclonal Antibody

品牌:solarbio | 货号:K007646P

英文名称 Anti-TFAP2B Polyclonal Antibody
宿主 Rabbit
别名 AP-2B;AP2-B
应用 WB
稀释比例 WB 1:200-1000.
交叉反应 Human Mouse
蛋白分子量 50kDa
Gene ID 7021
保存 Store at -20°C. Avoid freeze / thaw cycles.
储存液 Buffer: PBS with 0.03% Proclin300, 50% glycerol, pH7.3.
纯化方法 Affinity purification
亚型 IgG
免疫原 A synthetic peptide of human TFAP2B
性状 液体
Public Immunogen Range A synthetic peptide of human TFAP2B
Swiss Prot Q92481
克隆类型 Polyclonal Antibody
背景资料 This gene encodes a member of the AP-2 family of transcription factors. AP-2 proteins form homo- or hetero-dimers with other AP-2 family members and bind specific DNA sequences. They are thought to stimulate cell proliferation and suppress terminal differentiation of specific cell types during embryonic development. Specific AP-2 family members differ in their expression patterns and binding affinity for different promoters. This protein functions as both a transcriptional activator and repressor. Mutations in this gene result in autosomal dominant Char syndrome, suggesting that this gene functions in the differentiation of neural crest cell derivatives.

Anti-APOH Polyclonal Antibody-一抗-抗体-蛋白与免疫

Anti-APOH Polyclonal Antibody

品牌:solarbio | 货号:K008108P

英文名称 Anti-APOH Polyclonal Antibody
宿主 Rabbit
别名 BG;B2G1;B2GP1
应用 WB IHC
稀释比例 WB 1:500-2000. IHC 1:25-100.
交叉反应 Human
蛋白分子量 38kDa
Gene ID 350
保存 Store at -20°C. Avoid freeze / thaw cycles.
储存液 Buffer: PBS with 0.03% Proclin300, 50% glycerol, pH7.3.
纯化方法 Affinity purification
亚型 IgG
免疫原 Recombinant protein of human APOH
性状 液体
Public Immunogen Range Recombinant protein of human APOH
Subcellular Locations Cytoplasm
Swiss Prot P02749
克隆类型 Polyclonal Antibody
背景资料 Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. APOH may be a required cofactor for anionic phospholipid binding by the antiphospholipid autoantibodies found in sera of many patients with lupus and primary antiphospholipid syndrome, but it does not seem to be required for the reactivity of antiphospholipid autoantibodies associated with infections.?